Researchers writing in the Annual Review of Biochemistry have been investigating SOD1—superoxide dismutase 1 – an internally made enzymatic antioxidant and ALS. When the human body is biologically stressed SOD-1 is produced from copper-zinc precursors. Somehow in the development of ALS zinc-copper SOD1 becomes toxic, destroying motor neurons (nerves used for movement).
In a more recent report published in Neurobiology of Disease we learn that the provision of copper lengthens the lifespan of ALS mice by 25%.
Then, apparently on the cusp of a cure, researcher Stephen Byer indicates a zinc/copper drug is being tested and winks his eye to say, if you read between the lines, that maybe ALS patients ought to be taking copper and zinc supplements.