Once they had their target in sight, the group looked at the interaction between IDR-1018 and the peptides. Sure enough, when the two interacted, the signal was destroyed. The cells were left to fend for themselves. Over time, the bacteria would stress out and start to overproduce the (p)ppGpp molecules yet IDR-1018 would prevent them from finding solace. The biofilm would disappear and the remaining bacteria would be left helpless against any antibiotic treatment regimen.
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